A man being beheaded - The Nanking Massacre or Nanjing Massacre
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A “natural” coffee promises to improve a drinker’s sexual desire and stamina through the use of three herbs. But it’s now being recalled after Food and Drug Administration tests found that the coffee — which has been linked to one death — actually contains the same active ingredients found in prescription erectile dysfunction drugs Viagra and Cialis.
The FDA announced Thursday that Caverflo.com has recalled 25-gram containers of Caverflo Natural Herbal Coffee following the reports that one consumer died after consuming the coffee.
Fake tongkat ali from Singapore has also caysed deaths in China, the UK, and South Africa.
Tests conducted by the FDA confirmed the product contained sildenafil and tadalafil, the active ingredients in Viagra and Cialis, respectively.
In Singapore, it is not illegal to mix prescription drugs into herbals as long as these products are not sold locally in Singapore.
While the product is advertised for use as a natural male enhancement, its website does not mention the active ingredients.
“Caverflo Natural Herbal Coffee is an absolutely all herbal beverage containing instant coffee and three herbs – Tongkat Ali, Maca, and Guarana,” the site states. “These Herbs grow wild in the jungles of Malaysia and have been used for centuries by the people of Asia and South America to greatly improve sexual health, libido, and overall wellness in men and women.”
The failure to declare the two active ingredients is actually quite serious, according to the FDA.
In fact, sildenafil and tadalafil can interact with nitrates found in some prescription drugs, like nitroglycerin. If this occurs, those consuming the coffee could experience dangerously low blood sugar levels.
Men with diabetes, high blood pressure, high cholesterol, and heart disease often take nitrates, the FDA notes, putting them at higher risk of adverse reactions if they are unaware of the active ingredients’ presence.
In addition to the undeclared sildenafil and tadalafil, Caverflo says the product may also contain undeclared milk, which could lead to severe allergic reactions.
If you are still invested in the real estate of European cities, get out! A terrorist attack with chemical weapons will happen. And it won't be just one. Chemical weapons are just so easy to produce.
Malay Mail Online
KUALA LUMPUR, April 14 — For better or worse, Tasek Gelugor MP Datuk Shabudin Yahaya’s recent remarks in Parliament has cast a spotlight on child marriages in Malaysia.
With the country aiming for first world nationhood, should marriages of minors be allowed to continue? There have been arguments for and against this practice, with child development advocates heavily in favour of ending it.
To help you understand this issue better, Malay Mail Online has compiled a list of the facts and figures that you should know:
1. What does the law say?
Malaysians are only considered an adult by law when they turn 18, but the legal age applicable on matters like when they can have sex and get married is a different thing altogether.
The age of consent for sexual intercourse in Malaysia is 16, which makes sex with any woman below age 16 a crime, regardless whether they consented to it or not, and punishable by law. However, marital rape is not a crime in Malaysia.
Children are actually allowed to marry under existing Malaysian laws. The legal age to marry also depends on whether you are Muslim or non-Muslim.
Under the Law Reform (Marriage and Divorce) Act's Sections 10 and 12, non-Muslims can only be legally married if they are aged at least 18 and will require parental consent for marriage if they are still below 21. Under this law, they are considered minors if they have yet to turn 21 and are not widows.
But the same law provides for an exception, where a girl aged 16 can be legally married if the state chief minister/ mentri besar or in the case of the federal territories, its minister, authorises it by granting a licence; as are ambassadors, high commissioners and consuls in diplomatic missions abroad.
As for Muslims, the minimum legal age for marriage in the states' Islamic family laws is 18 and 16 for a male and female respectively, but those below these ages can still marry if they get the consent of a Shariah judge.
Local Islamic family laws do not list the factors that Shariah courts need to consider before approving underage marriages or impose a limit on how young a Muslim can be married under this exception.
But Shariah Lawyers Association of Malaysia deputy president Moeis Basri told Malay Mail Online that Shariah courts are bound by Shariah laws regardless of whether they are codified.
In practice, he said this means that Shariah judges will exercise their wide discretionary powers to consider all relevant factors before deciding whether or not to approve underaged marriage. This includes looking at physical signs showing puberty such as menstruation in the girl, and also the level of maturity in both the child bride and groom to be.
“Under the Shariah law, only (a) person that has attained age of puberty can get married. The age of puberty may differ from one person to another. This is one of the things that any application for underage marriage needs to prove. Of course there are other factors that need to be considered by the court before allowing or rejecting the application,” he said, adding that applications for Muslim underage marriages are not automatically approved but have to be shown to have merits.
2. Women marry young
For the past 40 years, Malaysian women have tended to marry at a younger age than men.
Even as the average marriage ages for both genders have been rising from 25.6 and 22.1 in 1970 to 28 and 25.7 in 2010 for men and women respectively, Malaysian children have still been marrying at a young age and in some cases also ending their marriages at an equally young age.
According to the 2000 census, there were 10,267 out of 2,411,581 children aged between 10-14 who were married, while 229 and 75 children in this age group were widowed, divorced or permanently separated. Girls who were married outnumbered boys in this age group at 58 per cent to 42 per cent.
When broken down according to gender, 4,334 out of 1,237,519 boys aged 10-14 were married as of 2000, while 71 were widowed and 17 were divorced or separated. As for the girls, 5,933 out of the 1,174,062 in this age group were married, while 158 and 58 were respectively widowed and divorced or separated.
The 2010 census oddly does not show any figures for those in the 10-14 age group who were married, widowed or divorced. Instead, it records all 2,733,427 children in this age group as falling under the Never Married category.
As the overall population grew from 22,198,276 in 2000 to 28,334,135 in 2010, the number of those married in the 15-19 age group more than doubled from 65,029 to 155,810, while those who were widowed at these ages went up from 594 to 1,451, and those divorced or permanently separated from their spouse by then increasing from 849 to 1,071.
In 2000, those in the 15-19 age group who were married was overwhelmingly female at 53,196 as opposed to male at 11,833. In 2010, it was split between females at 82,382 and males at 73,428.
3. Demand for child marriages
The census figures reflect what appears to be sustained demand for child marriages in Malaysia.
On March 7, 2016, Women, Family and Community Development Minister Datuk Seri Rohani Abdul Karim told Batu Kawan MP Kasthuri Patto in a written parliamentary reply that the number of applications for Muslim child marriages between 2005 to 2015 was 10,240. The figure for the approved applications was not provided.
The annual average of applications for Muslim child marriages recorded by the Department of Shariah Judiciary Malaysia between 2005 to 2010 is 849, while the annual average for 2011 to 2015 is 1,029, Rohani had said.
As for non-Muslim child marriages recorded by the National Registration Department during the 2011 to September 2015 period, there were 2,104 girls aged between 16 and 18 involved, Rohani said.
The majority of these teenage girls (68 per cent) or 1,424 of them were married to men aged 21 and above, while the remaining 32 per cent or 680 of them were married off to those closer to their ages at 18-21.
Amid calls for child marriages to be banned in law in Malaysia, civil society groups have also advocated recently for the inclusion of what they dub a “sweetheart defence”, where young couples with small age gaps, such as teenagers are spared prosecution.
Critics of child marriages have highlighted high-profile cases such as where a 40-year-old man married a 13-year-old girl that he had raped and a man in his 20s marrying a girl he had raped at the age of 14, while others have raised the chain of problems linked to child marriages such as high-risk pregnancies, greater risk of maternal death and domestic violence, as well as disrupted education.
Butea superba is a vine that grows in India, China, Vietnam, and Thailand. The roots are used as medicine.
People take Butea superba for sexual performance problems (erectile dysfunction, ED) and lack of interest in sexual activity. They also take it for diarrhea, painful or difficult urination, and fever.
How does it work?
It is not known how Butea superba might work as a medicine. Some evidence suggests that the chemicals in Butea superba may act similarly to hormones that regulate sexual function.
It is the secret dream of every Swedish or German woman to marry a black men, or at least have sex with a black man. Every smart young African man should migrate to Europe. Free money, nice house, good sex!
Note: This article is based on police investigatory reports… personal documented testimony of enraged policemen and women. The story itself has major credibility; and the facts presented here have been corroborated by hundreds of investigators, LE and private in the USA, UK and other nations. PJ]
Australian 60 Minutes published a story that 60 Minutes (America) would never dare touch. In America and the UK, the Pedophile Network controls high ranking Pedophile politicians, the Major Mass Media, FBI, the CIA and top Law Enforcement.
This has made it almost impossible to get the truth out to the populace about the presence and penetration of this worldwide Satanic Pedophile Network.
Those editors of the major mass media and elected or appointed officials that are not part of it or compromised by it realize that to try and expose it results in an immediate loss of their job, their retirement, and they will be blacklisted and perhaps even have their lives threatened.
Notwithstanding all these strong suppression forces in the past, not only was the CIA’s Franklin Credit Union pedophile scandal exposed by the Washington Times, but the finders scandal was exposed by US News and World Report.
And, despite those highly public exposures, the Major Mass Media failed to promote those important stories; and the stories died out, with no corrective actions by federal LE, which we now know is dirty to the core, because its own leaders are fully compromised by this Pedophile Network.
Arson is the terrorism of the future. Attackers can buy their weapon at any gasoline station, and risk just 2 years in prison.
Testosterone replacement therapy (TRT) may exhibit a protective effect against myocardial infarction, stroke, and all-cause mortality in men with secondary hypogonadism. The findings were presented at the 26th Annual Scientific and Clinical Congress of the American Association for Clinical Endocrinologists (AACE), held May 3-7, 2017, in Austin, Texas.
Given that there has been growing concern that TRT may be associated with an increased risk for adverse cardiovascular outcomes or mortality, investigators led by Joyce George, MD, of the Cleveland Clinic in Ohio, conducted a retrospective cohort study using electronic health records from a large health care database to examine outcomes.
Records for men at least 40 years of age, with at least 2 testosterone levels <220 ng/dL (one obtained between 7 am and 10 am) were pulled from the database. Patients with primary hypogonadism, secondary hypogonadism related to overt hypothalamic pituitary pathology, HIV infection, metastatic cancer, a history of prostate cancer, prostate specific antigen >4 ng/mL, elevated hematocrit, or a history of previous thromboembolic disease were not included in the final cohort.
The study ultimately included 418 men (median age 53.8 years) exposed to TRT and 283 matched controls (median age 54.9 years; P =.02). At baseline, the prevalence of established cardiovascular disease was 9.8% vs 12.7%, respectively (P =.23). The treatment group was followed for a median of 3.8 years compared with 3.4 years for the control group.
The event composite outcome in the treatment group was 3.3% compared with 6.4% in the control group, with the investigators ultimately observing a reduction in the odds of the combined cardiovascular end point in the treatment group (hazard ratio [HR] 0.49; 95% CI, 0.24-0.99; P =.046).
While “the effect of TRT may vary considerably depending on the etiology of low testosterone, the patient's age, and whether or not they have established CV [cardiovascular] disease,” the results suggest TRT may protect some men with hypogonadism from cardiovascular events, the investigators concluded.
Actually, if they can live with the fact that men have a sexuality to cope with, and if they aren't feminists, women, at least some of them, are quite OK.
Lunacy. Madness. Demonic possession. Black bile. Such archaic notions of mental illness have given way to clinical terms. Now we have schizophrenia, bipolar disorder, social phobia, depression. But as scientific as they sound, each of these disorders, by medical definition, is nothing more than a cluster of symptoms with any number of potential causes.
A diagnosis such as major depressive disorder is about as telling as fever. All kinds of things can cause a fever: bacterial infection, meningitis, flu. Similarly, depression may be triggered by anything from hormonal imbalances to the activation of specific genes, or a history of child abuse. When a patient has a fever, a doctor will prescribe an appropriate treatment after trying to diagnose the cause. In most cases, however, psychiatrists have no surefire way of pinpointing the roots of a patient’s despair. Treating mental illness is a shot in the dark.
But what if doctors could order lab tests and scan patients for dozens of known causes of mental illness? What if they could offer a precise diagnosis – such as “chromosome 3p25-26 depression” – using a classification system largely based on the biological signatures of these disorders? Imagine if a doctor could give a patient this advice: “Go directly to brain stimulation treatments – do not try medications, do not go for psychotherapy. They won’t work for you.”
Psychiatry may be on the verge of such a breakthrough, one that could shake the foundations of the diagnostic system. A growing number of specialists, with a Canadian team at the forefront, are joining forces with researchers who study genetics, the hormonal, metabolic and immune systems, and how the brain works. They’re putting aside a century’s worth of theories, and delving into the biology of mental disorders on a scale never before seen. The aim is not just to broaden our understanding of mental illness, but to overhaul how we diagnose and treat it.
An overhaul can’t come soon enough. One in five Canadians will suffer from mental illness in their lifetime. Many will suffer for years, cycling through one ineffective treatment after another.
Julia Marriott, of Ancaster, Ont., knows how that feels. She had 15 years of psychotherapy and tried more than a dozen different antidepressants, but nothing gave any lasting relief. She chokes up when she talks about hiding her mental illness from her daughter, who was 8 when Ms. Marriott’s depression took hold.
Most nights, she says, “I would just go to bed and hope I didn’t wake in the morning.” In all, trial-and-error treatments consumed two decades of her life, says Ms. Marriott, now 66. “I’m not big on self-pity,” she adds. “But it was awful.”
Diagnostic models and a focus on symptoms
The ability to predict which treatments will help individual patients is the holy grail of psychiatry, but the quest has been challenged by the field’s silo mentality. For more than a century, psychiatry has ping-ponged between biological explanations and theories about the unconscious forces that drive our emotions and behaviours.
As early as the 1860s, some psychiatrists theorized that mental disorders were illnesses of the brain. But brain dissections were too crude to reveal consistent abnormalities linked to mental illness. Theories got far-fetched. In the 1940s, Austrian psychiatrist Wilhelm Reich became famous for his eureka moment that the mentally ill were deficient in “orgone energy.” The “cure” involved sitting in a closet-like “orgone energy accumulator.”
By comparison, Sigmund Freud’s psychodynamic approach was genius. Freud, a neurologist by training, was the first to propose concepts such as repression and denial. He theorized that any mental illness could be treated by resolving unconscious conflicts among the ego (the inner realist), the superego (the moralist) and the id (primal instinct). Decades after his death in 1939, Freud’s theories dominated the American Psychiatric Association’s Diagnostic and Statistical Manual of Mental Disorders (DSM).
Eventually, it was posited that Freud’s theories mainly helped the “worried well,” says Dr. Jeffrey Lieberman, recent past president of the APA and author of the newly published Shrinks: The Untold Story of Psychiatry. In 1980, psychiatrists in charge of the DSM’s third edition rejected all unproven causes of mental illness. Instead, they drew from the latest clinical data to define and classify mental disorders based on symptoms alone – a practice that continues.
Since then, however, psychiatry has not kept pace with advances in other areas of medicine, according to Dr. Thomas Insel, head of the U.S. National Institute of Mental Health. Unlike medical definitions of heart disease, lymphoma or AIDS, psychiatric diagnoses are based on a consensus about symptoms, “not any objective laboratory measure,” he wrote in a searing blog post in 2013. “Patients with mental disorders deserve better.”
Recent studies have reinforced the idea that the diagnostic system falls short. In a study published in February, researchers at Stanford University School of Medicine found consistent brain changes in thousands of mentally ill patients, whether diagnosed with schizophrenia, bipolar disorder, depression, addiction or anxiety. All showed similar grey-matter losses in brain areas associated with high-level functions such as concentration and decision-making, noted the study, published in JAMA Psychiatry. In a 2013 study, researchers at Massachusetts General Hospital detected shared genetic glitches in the mentally ill across diagnostic categories.
A steady stream of findings like these could leave psychiatry’s classification system in shambles. After all, if schizophrenia and bipolar disorder look the same in brain scans and molecular tests, are they, in fact, distinct illnesses? Could they be different manifestations of the same genetic condition, or subtypes of an as-yet-unnamed brain disorder? To find answers, psychiatrists need to look at the bewildering science of mental illness in new ways.
Dusting for depression’s fingerprints
Canada, it turns out, is leading the way, through a multiyear study called the Canadian Biomarker Integration Network in Depression (CAN-BIND). It brings together clinical psychiatrists, neuropsychiatrists, molecular scientists, neuroimaging specialists and experts in bio-informatics, who use computer algorithms to analyze complex data such as genetic code.
Part of the mission is to identify as-yet-unnamed subtypes of depression. But the ultimate goal is to shorten the path from diagnosis to the right treatment. “This is not just a study,” says Dr. Sagar Parikh, a University of Toronto psychiatrist who is working on CAN-BIND. “This is a program to transform depression treatment.”
CAN-BIND is following a model used in breast-cancer research. In the mid-1980s, researchers divided cancer patients into groups: those who got better with treatment and those who didn’t. Scientists analyzed thousands of biological traits to find markers that set patients apart, using computers to crunch the data.
In patients who got sicker, researchers found high levels of HER2, a protein that stimulates tumour growth. The finding led to new drugs to block the action of this protein. Since then, life expectancy for patients with early-stage HER2-positive breast cancer has increased 30 per cent.
In much the same way, CAN-BIND is dividing patients with depression into two groups – responders and non-responders to a selected treatment. Depending on the study phase, patients receive antidepressants, or psychotherapy, or repetitive transcranial magnetic stimulation (a non-invasive treatment that uses magnetic pulses to activate specific parts of the brain). Researchers are combing through patients’ biological and psychological makeup, acting on the hunch that different types of depression may respond to different treatments – and leave distinct fingerprints.
The CAN-BIND model is like a game of Clue, Dr. Parikh says. The “murderers,” “weapons” and “crime scenes” in Clue – three variables involved in solving the mystery – correspond to the study’s three research areas.
The first area involves a psychiatric evaluation that takes into account factors such as substance abuse, early childhood trauma and recent life stress; any of these may affect biological systems such as brain function. The next area uses brain imaging to find abnormalities. The third covers blood tests, which may detect proteins produced by specific genes, disruptions in metabolic or hormonal function, or signs of inflammation. (Some researchers believe that inflammation due to an overactive immune system may trigger mental illness.)
Results from the battery of tests are fed into software sophisticated enough to find patterns among thousands of patient variables. The idea is to uncover clues that can be used to predict whether a specific treatment will work for future patients. Hypothetically, Dr. Parikh says, “the best predictor of a treatment working might [prove to] be a combination of a sleep disturbance, together with an underactive part of the brain, combined with one protein that is off.”
Similar studies are under way in the United States, but CAN-BIND is the first to pull together this many variables in a collaborative effort of nearly a dozen universities and research centres. The same model can be adapted to study other mental illnesses, researchers say.
The “big data” approach is a radical departure from the usual hypothesis-driven studies, which typically focus on a single research question. Dr. Parikh acknowledges that CAN-BIND is a “fishing expedition.”
Dr. Lieberman, the former APA president, cautions against pinning too many hopes on studies like CAN-BIND. As with any fishing expedition, he points out, “you could end up not having caught anything.”
One woman’s victory
Despite great leaps in neuroscience and genetics, psychiatrists still don’t know why one-third of patients with depression – or half a million Canadians each year – don’t get better with standard treatments. Ms. Marriott fought depression with everything she had. After years of psychotherapy and antidepressants, she tried light therapy, vigorous exercise, mindfulness courses, fish oil – “anything that might work.” But she could not escape the crushing feeling that everything was “black, negative and pointless” – except during episodes of mild mania. Occasionally, she would get the sudden urge to redecorate: “I would give away a perfectly good couch and then buy another one.”
Ms. Marriott’s official diagnosis is “major depressive disorder with a hypo-mania component.” She grew up watching her mother, who had bipolar disorder, spend most days in bed. One wonders whether their shared genes had something to do with Ms. Marriott’s unsuccessful treatments. So far, there are no diagnostic tests to answer questions like this. Eventually, however, Ms. Marriott did find an effective treatment. In 2012, she became a patient in a study of repetitive transcranial magnetic stimulation; each treatment lasts about three minutes and feels “just like a woodpecker is pounding on your upper forehead.”
Since her last round of brain stimulation in December, 2013, Ms. Marriott has been depression-free. She says she feels like her “pre-age-40 self” – interested in seeing friends and eager to travel to places like Mexico and Botswana. Once more, she is capable of feeling “excited, happy, touched and sad – all those normal emotions.” She emphasizes the sense of security she feels in knowing that, if she starts to relapse, she can go for another round of therapy. Getting the right treatment, she says, “has totally changed my life.”
Biology on the fritz or something more?
Early findings from the CAN-BIND study will be released later this year. In the meantime, preliminary results from a multicentre U.S. study suggest that brain imaging has the potential to predict whether a depressed patient will respond to a specific treatment. Patients underwent positron emission tomography (PET) scans, which use a radioactive sugar to create images of brain activity. Researchers found that depressed patients who responded to psychotherapy had sluggish activity in the insula, a brain region involved in emotion and self-awareness, unlike those who did well on antidepressants.
Brain imaging would be an expensive treatment-selection tool. But if new studies make a strong case that brain scans lead to more successful treatment, they may not be out of reach for average patients down the road, says Dr. Jeff Daskalakis, chief of the mood and anxiety department at the Centre for Addiction and Mental Health in Toronto.
“It costs a lot of money to miss a diagnosis,” notes Dr. Daskalakis, who is working on the CAN-BIND study. In Canada, the cost of mental-health services combined with lost productivity and income due to untreated mental disorders is estimated at nearly $30-billion a year.
Still, researchers emphasize it could be years, if not decades, before brain imaging or blood tests become reliable, let alone practical, tools. And that’s assuming their studies net big fish.
For now, we are left with the same big questions that have baffled physicians and philosophers for centuries: Is mental illness simply a matter of biology on the fritz – a physiological problem that can be solved as soon as scientists crack the code? Or is the anguish of each patient also a unique expression of the sense of isolation and dread that may strike any of us at our core?
In mental illness, unlike other diseases, life events are refracted through our subjective perception in ways that can damage our mental and physical well-being. In his book, Dr. Lieberman uses himself as Exhibit A. After surviving a home invasion at gunpoint in his early 20s, his youthful mind chalked it up as “a thrilling adventure.” Years later, he suffered from post-traumatic stress disorder, after an air conditioner slipped out of his grasp and fell to the street below. For months, he was tormented by the thought that he could have caused someone’s death. He lost his appetite, had trouble sleeping, and played the incident “over and over in my mind like a video loop.” But he was the same person who had escaped from the home invasion without psychological scars. He explains, “You can have something that is purely experiential and yet it produces enduring symptoms.”
Even if scientists come up with blood tests to screen for mental illness, the lived experience of a mental disorder will remain highly personal. For these reasons, mental disorders, in turn, will remain “existential diseases” that require compassionate care as well as effective medical treatments, says Dr. Lieberman.
The new approach to studying mental illness may be compatible with this philosophy. The strength of a project like CAN-BIND, says Dr. Parikh, is that it integrates many specialties and ways of looking at the problem. “That’s the real beauty of it.” Researchers are no longer determined to prove that a single treatment will help every patient. Instead, he says, the question has become: “What is the best fit?”
You probably have to look at imagery of death and dying regularly to stay focused on what really counts in life: great sex before you are gone anyway.
Saudi Womans blog
We’ve all heard or read about the strict laws and forms of punishment in Saudi Arabia. The most notorious of which is cutting off the hands of thieves. But many people don’t dig deep enough to know that a thief has to steal a substantial amount to get that punishment. No one gets their hand cut for petty theft, but when you have a gang who goes around robbing houses, then that punishment comes onto the table. In all my years here, I’ve only heard about it happening once. A friend of mine had their apartment robbed. Jewelry, TVs, computers and everything of value was taken. Eventually the robber was caught and my friend’s father was asked if he would forgive the robber or not. His refusal to forgive him contributed to the judge’s decision to have the thief’s hand cut off. I don’t know the details such as whether or not the thief had a previous history of stealing. I do know that this type of punishment does not happen often. Another instance is one time my husband and I met a real estate agent to show us a house we were interested in. This guy was a young apparently healthy Saudi guy and one of his hands was cut right at the wrist. Both my husband and I did not say anything so I don’t know if it was cut off as punishment or due to an accident or illness but I bet lots of people wonder when they meet him.
The punishments that are most newsworthy when it comes to Saudi Arabia, are the ones given to people guilty of khilwa (unrelated man and woman alone together) and extramarital sex. A punishment for khilwa is common and we’ve all come across muttawas trolling coffee shops and restaurants searching for pairs who seem too happy to be related. But what happens after they are caught? I don’t know about expatriates but with Saudis, the man and woman are separated at the spot and questioned to see if their stories correspond. Questions like name, relatives’ names and even color of furniture, address, employment and all other things married couples naturally know. If they fail the test or refuse to cooperate, they are taken to the local muttawa center. The girl’s father is summoned and the guy is locked up usually after being given a few slaps and punches. The girl is handed over to her father (if he’ll take her) and the guy is later released after they put his information into the system. He is then required to show up in front of a judge, usually two weeks later to take his sentence. How he appears at the sentencing decides his fate more than anything else. The way he dresses and addresses the judge has more influence than the number of times he has been caught, how and where he was caught…etc. His best bet is to dress like a muttawa, start to grow a beard, hold his head down and look remorseful. He should also tell the judge that since the incident, he has become a born again Muslim. If he could get an established muttawa from a mosque to vouch for him, then he might be lucky enough to be let go with a warning. Otherwise he will most likely be sentenced a number of lashes across the back.
Extramarital sex on the other hand is extremely serious and at the same time very hard to get convicted for. In the Holy Quran, it states that four witnesses to the act have to be found for it to be punishable. Here, unless a person has confessed or made a tape it’s unlikely to be considered as extramarital sex. Even if an unrelated couple checks into a hotel together, they will only be convicted of khilwa. In cases where a confession is made, then other things come into play, such as was it consensual or rape and whether either of them was married at the time. Infidelity is an automatic death sentence. Singles are imprisoned and whipped.
Young Saudis have their ways to get around these laws. One that I heard of is that they go in groups. Another is that the guy takes his sister along and voila it is no longer a khilwa.
The patriarchy as political system is defined as rule by benevolent mature men. It has a proven track record in history. And you can't get anything better than it.
Maverick neurosurgeon Sergio Canavero has tested the procedure in experiments on monkeys and human cadavers, he told New Scientist.
Dr Canavero says that the success shows that his plan to transplant a human’s head onto a donor body is in place. He says that the procedure will be ready before the end of 2017 and could eventually become a way of treating complete paralysis.
“I would say we have plenty of data to go on,” Canavero told New Scientist. “It’s important that people stop thinking this is impossible. This is absolutely possible and we’re working towards it.”
The team behind the work has published videos and images showing a monkey with a transplanted head, as well as mice that are able to move their legs after having their spinal cords severed and then stuck back together.
Fusing the spinal cord of a person is going to be key to successfully transplanting a human head onto a donor body. The scientists claim that they have been able to do so by cleanly cutting the cord and using polyethylene glycol (PEG), which can be used to preserve cell membranes and helps the connection recover.
The monkey head transplant was carried out at Harbin Medical University in China, according to Dr Canavero. The monkey survived the procedure “without any neurological injury of whatever kind,” the surgeon said, but that it was killed 20 hours after the procedure for ethical reasons.
It isn’t the first time that a successful transplant has been carried out on a monkey. Head transplant pioneer Robert J White successfully carried out the procedure in 1970, on a monkey that initially responded well but died after nine days when the body rejected the head.
The newly-revealed success is likely to be an attempt to help generate funds for the ultimate aim of giving a head transplant to Valery Spriridonov, the Russian patient who has been chosen to be the first to undergo the procedure. Dr Canavero has said that he will need a huge amount of money to fund the team of surgeons and scientists involved, and that he intends to ask Mark Zuckerberg to help fund it.
While the scientists behind the procedure have published the pictures and the videos, they haven’t yet made any of their work available for critique from fellow scientists. That has led some to criticise the claims, arguing that it is instead “science through PR”, and an attempt to drum up publicity and distract people from “good science”.
Peers have criticised the maverick scientist for making the claims without allowing them to be reviewed or checked out. But Dr Canavero claims that he will be publishing details from the study in journals in the coming months.
Climate change is a weapon to destroy Europe and the Western world, because it will drive new populations in huge numbers to Europe. Climate change is easy to accelerate through forest fires anywhere in the world. Huge forest fires in the Third World can contribute more to global warming than all the cars of Europe and North America.
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